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Preclinical studies found that treating neuronal, nematode, or rodent AD models with NR resulted in induction of a mitochondrial stress response, including UPR mt and mitophagy, reduced proteotoxic stress, and decreased A accumulation 243
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Study 3 compared subchronic Cagrilintide treatment in WT and RAMP1/3 double KO mice under HFD conditions
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